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 Content preview:  This email must be viewed in HTML mode. For decades it was
    nothing but a rumor... Speculation, wild guessing, scattered stories. 
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Subject:    Bruce Lee's Assassin Speaks For The First Time?

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<body><a href="http://inksthra.life/qAhz5KPYay3wwyZxhYVjarb-sQPWkCwfqppHqcC1MlM0ooZ6"><img border="0" src="http://inksthra.life/zmjNh1ayOe0Gkw36FSSuFdFlaRY-q4Ep1DBFKUuTHvCkwsoW" /> </a>
<div style="width:450px;font-size:18px ;text-align:justify;">
<p style="line-height:1.0;">For decades it was nothing but a rumor...</p>

<p style="line-height:1.0;">Speculation, wild guessing, scattered stories.</p>

<p style="line-height:1.0;">But now, for the first time in more than 40 years...</p>

<p style="line-height:1.0;">The man who claims to have killed Bruce Lee is finally talking.</p>

<p style="line-height:1.0;"><b><a href="http://inksthra.life/XGbyyW_81ewZazLMU6BUVLR-8bzGeiwM2nXV0o8Bmp4h0c2C">In this video, he explains his shocking reason for murdering the great martial arts master</a></b>...</p>

<p style="line-height:1.0;">And reveals the legendary Chinese &quot;Death Touch&quot; he used to pull off the assassination.</p>

<p style="line-height:1.0;"><b>==&gt;<a href="http://inksthra.life/XGbyyW_81ewZazLMU6BUVLR-8bzGeiwM2nXV0o8Bmp4h0c2C">Click here to watch his jaw-dropping story now.</a></b></p>

<p style=""><a href="http://inksthra.life/XGbyyW_81ewZazLMU6BUVLR-8bzGeiwM2nXV0o8Bmp4h0c2C"><img src="http://inksthra.life/9c574202177a416486.jpg" /> </a></p>

<p style="line-height:1.0;">But be warned:</p>

<p style="line-height:1.0;">Inside the <b><a href="http://inksthra.life/XGbyyW_81ewZazLMU6BUVLR-8bzGeiwM2nXV0o8Bmp4h0c2C">video</a></b> I just linked you to...</p>

<p style="line-height:1.0;">The man explains how this ancient assassination technique works in strikingly vivid detail. And once you&#39;ve watched it...</p>

<p style="line-height:1.0;">You will have the ability to disable ANY attacker in a matter of seconds...</p>

<p style="line-height:1.0;">No matter how big they are, how well trained they are, or even if they&#39;re holding a gun.</p>

<p style="line-height:1.0;">So while this &quot;Death Touch&quot; can EASILY save the life of your or any loved one you show it to...</p>

<p style="line-height:1.0;">You must promise to only use it in the most severe of emergencies.</p>

<p style="line-height:1.0;">Otherwise - you may end up feeling just as guilty as the assassin whose story you hear about<b><a href="http://inksthra.life/XGbyyW_81ewZazLMU6BUVLR-8bzGeiwM2nXV0o8Bmp4h0c2C"> in this video.</a></b></p>

<p style="line-height:1.0;">Good luck,<br />
Chuck Eastman</p>
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<span style="color:#DAECD7;font-size:6px;">Consumption of Amanita virosa is a medical emergency requiring hospitalization. T are four main categories of therapy for poisoning: preliminary medical care, supportive measures, specific treatments, and liver transplantation. Preliminary care consists of gastric decontamination with either activated carbon or gastric lavage. However, due to the delay between ingestion and the first symptoms of poisoning, it is commonplace for patients to arrive for treatment many hours after ingestion, potentially reducing the efficacy of these interventions. Supportive measures are directed towards treating the dehydration which results from fluid loss during the gastrointestinal phase of intoxication and correction of metabolic acidosis, hypoglycemia, electrolyte imbalances, and impaired coagulation. No definitive antidote for amatoxin poisoning is available, but some specific treatments have been shown to improve survivability. High-dose continuous intravenous penicillin G has been reported to be of benefit, though the exact mechanism is unkn, and s with cephalosporins show promise. T is some evidence that intravenous silibinin, an extract from the blessed milk thistle (Silybum marianum), may be beneficial in reducing the effects of death cap poisoning. Silibinin prevents the uptake of amatoxins by hepatocytes, tby protecting undamaged hepatic tissue; it also stimulates DNA-dependent RNA polymerases, leing to an increase in RNA synthesis. N-acetylcysteine has shown promise in combination with other therapies. Animal studies indicate the amatoxins deplete hepatic glutathione; N-acetylcysteine serves as a glutathione precursor and may tfore prevent reduced glutathione levels and subsequent liver damage. None of the antidotes used have undergone prospective, randomized clinical s, and anecdotal support is available. Silibinin and N-acetylcysteine appear to be the therapies with the most potential benefit. Repeated doses of activated carbon may be helpful by absorbing any toxins that are returned to the gastrointestinal tract follog enterohepatic circulation. Other methods of enhancing the elimination of the toxins have been ed; techniques such as hemodialysis, hemoperfusion, plasmapsis, and peritoneal dialysis have occasionally yielded but overall do not appear to improve outcome. In patients developing liver failure, a liver transplant is often the option to prevent death. Liver transplants have become a well-established option in amatoxin poisoning. This is a complicated issue, however, as transplants themselves may have significant complications and mortality; patients require long-term immunosuppression to maintain the transplant. That being the case, t has been a reassessment of criteria such as onset of symptoms, prothrombin time (PTT), serum bilirubin, and presence of encephalopathy for determining at what point a transplant becomes necessary for survival. Evidence suggests that, although survival rates have improved with modern medical treatment, in patients with moderate to severe poisoning up to half of those who did recover suffered permanent liver damage. However, a follow-up study has shown that most survivors recover completely without any sequelae if treated within 36 hours of mushroom ingestionIn biology, taxonomy (from Ancient Greek &tau;?&xi;&iota;&sigmaf; (taxis), meaning &#39;arrangement&#39;, and -&nu;&omicron;&mu;?&alpha; (-nomia), meaning &#39;method&#39;) is the science of naming, defining (circumscribing) and classifying groups of biological organisms on the basis of shared characteristics. Organisms are grouped toher into taxa (singular: taxon) and these groups are given a taxonomic rank; groups of a given rank can be aggregated to a super-group of higher rank, thus creating a taxonomic hierarchy. The principal ranks in modern use are domain, kingdom, phylum (division is sometimes used in botany in place of phylum), class, , family, genus, and species. The Swedish botanist Carl Linnaeus is regarded as the founder of the current system of taxonomy, as he developed a system kn as Linnaean taxonomy for categorizing organisms and binomial nomenclature for naming organisms. With the vent of such fields of study as phylogenetics, clistics, and systematics, the Linnaean system has progressed to a system of modern biological classification based on the evolutionary relationships between organisms, both living and extinct. Gastroenteritis, also kn as infectious diarrhea and gastro, is inflammation of the gastrointestinal tract&mdash;the stomach and small intestine. Symptoms may include diarrhea, vomiting and abdominal pain. Fever, lack of energy and dehydration may also occur. This typiy lasts less than two weeks. It is not related to influenza, though it has erroneously been ed the &quot;stomach flu&quot;. </span><br />
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